Chris McAtomney

Pizza isn’t junk food, and a burger might not be, either. 

A donut is. So are chocolate, lollies, and chips.

Same calorie density, completely different nutrient density. The question shouldn’t be “is this food bad” - it should be “what am I getting for those calories, and is my energy expenditure sufficient to offset them?”


Before you begin a parasite detox, get your ferritin checked. Fatigue, brain fog, mood swings, interrupted sleep — the symptoms people often pin on “parasites” are the exact symptoms of iron deficiency, the most common nutrient deficiency on the planet.

Occam’s razor: rule out the boring, likely thing before you chase the exotic one. And if you’re getting infusions “every now and then,” that’s a sign nobody’s asked why your iron keeps dropping.

We’ve known for a while that opioids lower serum testosterone, which is probably of little consequence, in that I don’t imagine raising serum testosterone is a high priority for most he**in users 😉

But perhaps lesser known is that benzos also significantly lower testosterone by inhibiting hypothalmic release of GnRh. So if you’re in the habit of using Va**um or Xanax to alleviate anxiety or induce sleep, you might be inadvertently lowering your T, too.

A lot of what we call luck is shaped by how people frame their setbacks.

Some people turn challenges into learning opportunities, so it seems like they just keep winning.

It’s not that nothing adverse ever afflicts them. It’s that they don’t let these events define them.

Feeling tempted to revert back to your previous build? Don’t think about your why. In fact, don’t think about it at all.

You’ve already been over this during the planning stage. You know the why. It requires no further analysis.

Right now, your best option is to embody the robot.
The OS update has already been installed.

This is the path to building healthy behaviours independent of motivation. And that, is quietly powerful.

For a long time, liver fibrosis in MASH has largely been viewed as something we could only slow, rather than reverse. This study suggests that may not always be the case.

Semaglutide isn’t a substitute for addressing the underlying drivers of metabolic disease, and we still need longer-term safety and outcome data.

But it’s exciting to see collaborative research efforts yield these kind of results, and another reminder that improving metabolic health can have profound effects on all organs and systems, well beyond the number on the scales.

If you’d like to read the study yourself:
https://pubmed.ncbi.nlm.nih.gov/40305708/

You don’t need a gym buddy.

Red meat consumption is commonly perceived as being a significant risk factor for chronic disease and colorectal cancer.

The reality is that much of that risk has a lot to do with what kind of red meat, how you cook it, what you eat it with, and your overall fitness profile and inflammation status.

And if you get savvy about these nuances, you can significantly skew the risk:benefit ratio in your favour.

The most interesting insight that my CGM experiment yielded was that CGMs aren’t very useful for people without diabetes.

I’d hoped to identify which foods, training styles, supplements caused pronounced deviations outside my fasting blood glucose levels. And it turns out, as you’d expect- oats, potatoes, bread, all caused a rise. But being extremely insulin and glucagon sensitive, any deviation was rapidly corrected.

The only other standout was that my BGL regulation was a fraction looser after a night of restless sleep (as expected).

The key takeaway?

Unless you have diabetes, you don’t need a device to tell you what’s causing insulin resistance. We already know what drives it- poor sleep, sedentarism, unmanaged stress, and excess consumption of calorie dense high GI foods.