07/05/2026
The only thing we LOVE more than testing you good people is NEW KIT DAY.
Can't wait to put our new COSMED QNRG through it's paces to deliver even more high quality data and insight to our Performance Testing Services.
Go to
www.athletelab.uk
To learn more
13/04/2026
Just a selection of our hundreds of tests performed so far across our 3 London locations.
WE DON'T JUST DO RUNNING
We can test you on:
- Incline walking
- Rowing
- Cycling (including using your own bike set-up)
Vo2max, lactate, body composition, nutrition, strength, power, blood work and so much more.
Www.athletelab.uk
01/04/2026
He trained for months. He'd been tested. He still fell apart.๐จ
And here's what makes this case particularly important โ The methodology was sound in principle.
But the analyser was inaccurate. And critically, there was no cross-validation โ no concurrent lactate profiling, no comparison against performance metrics, no sense-check against training RPE or race history.
There's a further point worth making. To an experienced practitioner, the numbers themselves should have raised questions. VT1 was identified at an intensity unusually high for an athlete of his training background โ the kind of value that prompts a second look, a cross-reference with lactate, a conversation about training history. That scrutiny never happened. The data was accepted, the zones were issued, and the athlete had no reason to doubt either.
This is what separates rigorous testing from testing that merely looks rigorous.
But you can only catch that discrepancy if you have the experience to know what the numbers should look like in the first place.
Good testing isn't just about having a methodology or the 'kit'. It's about understanding the data and how to use it correctly.
Data + insight = success
๐ athletelab.uk
24/03/2026
The heart or the muscle โ what actually limits your endurance? ๐ซ๐ฆต
This is one of the most debated questions in exercise physiology. And a 2021 review by van der Zwaard, Brocherie & Jaspers gives us the most complete answer yet โ by going all the way down to the muscle fibre level.
Here's the honest answer: it's both, but they operate at different levels.
The heart sets the ceiling.
Classic one-legged exercise studies show the muscle can consume far more oxygen per unit mass when the heart isn't the bottleneck. Extra red blood cells and hyperoxic breathing both raise VOโmax โ confirming that cardiac output is the primary limiter of absolute VOโmax.
The cardiovascular argument is strong.
But the muscle determines how close to that ceiling you race.
And this is where it gets interesting. Skeletal muscle characteristics alone explained 67% of the variance in performance VOโ in elite cyclists. Mitochondrial oxidative capacity accounts for ~90% of VOโmax during cycling. Capillarisation + lactate threshold explained 92% of time-to-fatigue variance. In elite athlete groups โ where cardiac outputs are similarly high โ peripheral muscle factors become the decisive differentiator.
The key muscle-level players:
โ Type I fibre proportion โ more efficient at endurance frequencies, directly linked to economy
โ Mitochondrial density โ scales proportionally with VOโmax across cardiac patients to pro cyclists
โ Capillarisation โ up to 9 capillaries per fibre in elite cyclists vs 3โ4 in untrained. The bridge between cardiac delivery and mitochondrial uptake
โ Myoglobin โ the underappreciated Oโ transporter inside the fibre, ~50% higher in Type I vs Type II
The practical synthesis: train your heart AND your muscle. Polarised training does both โ long low-intensity work drives peripheral adaptations, targeted high-intensity blocks protect and develop cardiac function. Neither system operates in isolation.
Understanding which is your limiting factor is where physiology testing becomes genuinely useful.
Data + insight = success
๐ athletelab.uk